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Cation-independent M6PR (IGF2R) Rabbit mAb - A3762

Cation-independent M6PR (IGF2R) Rabbit mAb - A3762

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Cation-independent M6PR (IGF2R) Rabbit mAb

Sizes: 20µl, 100µl

Catalogue Numbers: A3762-20, A3762-100

Citations, Manuals and SDS Available upon request.

Background: This gene encodes a receptor for both insulin-like growth factor 2 and mannose 6-phosphate. The binding sites for each ligand are located on different segments of the protein. This receptor has various functions, including in the intracellular trafficking of lysosomal enzymes, the activation of transforming growth factor beta, and the degradation of insulin-like growth factor 2. Mutation or loss of heterozygosity of this gene has been association with risk of hepatocellular carcinoma. The orthologous mouse gene is imprinted and shows exclusive expression from the maternal allele; however, imprinting of the human gene may be polymorphic, as only a minority of individuals showed biased expression from the maternal allele (PMID:8267611).

Synonyms: MPR1; MPRI; CD222; CIMPR; M6P-R; MPR300; CI-M6PR; MPR 300; M6P/IGF2R; Cation-independent M6PR (IGF2R)

GeneID: 3482

Swiss-Prot: P11717

Reactivity: Human, Mouse, Rat

Calculated Molecular Weight: 274kDa

Observed Molecular Weight: 274kDa

Immunogen: A synthetic peptide corresponding to a sequence within amino acids 2392-2491 of human Cation-independent M6PR (IGF2R) (NP_000867.3).

Immunogen Species: Human

Host: Rabbit

Isotype: IgG

Purity: Affinity purification

Tested Applications: ELISA, WB, IF/ICC

Storage Buffer: Store at -20℃. Avoid freeze / thaw cycles. | Buffer: PBS with 0.02% sodium azide, 0.05% BSA, 50% glycerol, pH7.3.

Recommended Dilutions: WB: 1:500 - 1:1000 | IF/ICC: 1:50 - 1:200

Research Areas: Signal transduction, Immunology inflammation, CDs, Cell intrinsic innate immunity signaling pathway

Modifications: Unmodified

Conjugate: Unconjugated

Positive Samples: A-549, Mouse heart

Cellular Location: Lysosome Membrane, Single-pass Type I Membrane Protein

Category: Monoclonal Antibodies

Citations, Manuals and SDS Available upon request.